• http://rdf.wikipathways.org/Pathway/WP4549_r134295/Interaction/idd2e2d64e
  • http://vocabularies.wikipathways.org/gpml#hasComment
    • http://rdf.wikipathways.org/Pathway/WP4549_r134295/Comment/3759e98419850ad927159695f762fc56
• http://rdf.wikipathways.org/Pathway/WP4549_r134295/Comment/3759e98419850ad927159695f762fc56
  • http://www.w3.org/1999/02/22-rdf-syntax-ns#type
    • http://vocabularies.wikipathways.org/gpml#Comment
  • http://purl.org/dc/terms/isPartOf
    • http://rdf.wikipathways.org/Pathway/WP4549_r134295/Interaction/idd2e2d64e
  • http://vocabularies.wikipathways.org/gpml#commentText
    • AKT directly phosphorylates TSC2 on Ser939, Ser981 and Thr1462. This leads to inactivation of TSC2, which inhibits its GAP (GTPase-accelerating protein) activity