SPARQL | HTML5 RDFa and Microdata document 
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https://identifiers.org/mirbase/MI0001160   https://identifiers.org/mirbase/MI0001165   https://identifiers.org/mirbase/MI0001524   https://identifiers.org/mirbase/MI0001525   https://identifiers.org/mirbase/MI0003492   https://identifiers.org/mirbase/MI0003538   https://identifiers.org/mirbase/MI0004118   https://identifiers.org/mirbase/MI0005550   https://identifiers.org/mirbase/mmu-mir-128-2   https://identifiers.org/mirbase/mmu-mir-503   https://identifiers.org/pubmed/15651302   https://identifiers.org/pubmed/15952880   https://identifiers.org/pubmed/16022590   https://identifiers.org/pubmed/17911161   https://identifiers.org/pubmed/18426756   https://identifiers.org/wikipathways/WP3638   https://identifiers.org/wikipathways/WP723   https://identifiers.org/ensembl/ENSMUSG00000002068   https://identifiers.org/chebi/CHEBI:26523   https://identifiers.org/wikipathways/WP295   https://identifiers.org/ensembl/ENSMUSG00000004864   https://identifiers.org/ensembl/ENSMUSG00000020182   https://identifiers.org/ensembl/ENSMUSG00000022610   https://identifiers.org/ensembl/ENSMUSG00000025076   https://identifiers.org/ensembl/ENSMUSG00000027997   https://identifiers.org/ensembl/ENSMUSG00000028914   https://identifiers.org/ensembl/ENSMUSG00000029863   https://identifiers.org/ensembl/ENSMUSG00000031628   https://identifiers.org/ensembl/ENSMUSG00000053137   https://identifiers.org/ensembl/ENSMUSG00000053436   https://identifiers.org/kegg.compound/C00082   https://identifiers.org/kegg.compound/C00355   https://identifiers.org/kegg.compound/C03758   https://identifiers.org/wikipathways/WP493   https://identifiers.org/ensembl/ENSMUSG00000001924   https://identifiers.org/ensembl/ENSMUSG00000019505   https://identifiers.org/ensembl/ENSMUSG00000019979   https://identifiers.org/ensembl/ENSMUSG00000029223   https://identifiers.org/ensembl/ENSMUSG00000032596   https://identifiers.org/wikipathways/WP1254   http://rdf.wikipathways.org/Pathway/WP3638_r137774/Complex/b48c0   http://rdf.wikipathways.org/Pathway/WP3638_r137774/ComplexBinding/b48c0   http://rdf.wikipathways.org/Pathway/WP3638_r137774/WP/Interaction/f3d22   http://rdf.wikipathways.org/Pathway/WP3638_r137774/WP/Interaction/id15c6ffdf   http://rdf.wikipathways.org/Pathway/WP3638_r137774/WP/Interaction/id1687f4a7   http://rdf.wikipathways.org/Pathway/WP3638_r137774/WP/Interaction/id6655e7d4   http://rdf.wikipathways.org/Pathway/WP3638_r137774/WP/Interaction/id78636171   http://rdf.wikipathways.org/Pathway/WP3638_r137774/WP/Interaction/id982bbd29   http://rdf.wikipathways.org/Pathway/WP3638_r137774/WP/Interaction/id9fd46884   http://rdf.wikipathways.org/Pathway/WP3638_r137774/WP/Interaction/idb05a8a6d   http://rdf.wikipathways.org/Pathway/WP3638_r137774/WP/Interaction/idb1cc7404   http://rdf.wikipathways.org/Pathway/WP3638_r137774/WP/Interaction/idcd20a6ff   http://rdf.wikipathways.org/Pathway/WP3638_r137774/WP/Interaction/idd801024   http://rdf.wikipathways.org/Pathway/WP3638_r137774/WP/Interaction/idd9dda822   http://rdf.wikipathways.org/Pathway/WP3638_r137774/WP/Interaction/idebf7a170   http://rdf.wikipathways.org/Pathway/WP3638_r137774/WP/Interaction/idf9a647c5   https://identifiers.org/wikipathways/WP3638_r137774   
      http://www.w3.org/1999/02/22-rdf-syntax-ns#type   
          http://www.w3.org/2004/02/skos/core#Collection 
          http://vocabularies.wikipathways.org/wp#Pathway 
         http://vocabularies.wikipathways.org/wp#cellTypeOntologyTag   
          http://purl.obolibrary.org/obo/CL_0000540 
         http://vocabularies.wikipathways.org/wp#diseaseOntologyTag   
          http://purl.obolibrary.org/obo/DOID_12217 
          http://purl.obolibrary.org/obo/DOID_14330 
         http://vocabularies.wikipathways.org/wp#isAbout   
          http://rdf.wikipathways.org/Pathway/WP3638_r137774 
         http://vocabularies.wikipathways.org/wp#ontologyTag   
          http://purl.obolibrary.org/obo/DOID_12217 
          http://purl.obolibrary.org/obo/CL_0000540 
          http://purl.obolibrary.org/obo/DOID_14330 
          http://vocabularies.wikipathways.org/wp#Curation:AnalysisCollection 
          http://purl.obolibrary.org/obo/PW_0000014 
          http://purl.obolibrary.org/obo/PW_0000018 
          http://purl.obolibrary.org/obo/PW_0000013 
          http://vocabularies.wikipathways.org/wp#Curation:Diseases 
          http://vocabularies.wikipathways.org/wp#Curation:ExRNA 
         http://vocabularies.wikipathways.org/wp#organism   
          http://purl.obolibrary.org/obo/NCBITaxon_10090 
         http://vocabularies.wikipathways.org/wp#organismName   http://vocabularies.wikipathways.org/wp#pathwayOntologyTag   
          http://purl.obolibrary.org/obo/PW_0000014 
          http://purl.obolibrary.org/obo/PW_0000018 
          http://purl.obolibrary.org/obo/PW_0000013 
         http://xmlns.com/foaf/0.1/img   
          https://www.wikipathways.org//wpi/wpi.php?action=downloadFile&type=png&pwTitle=Pathway:WP3638&oldid=137774 
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         http://xmlns.com/foaf/0.1/page   
          http://www.wikipathways.org/instance/WP3638_r137774 
         http://purl.org/dc/elements/1.1/identifier   
          https://identifiers.org/wikipathways/WP3638 
         http://purl.org/dc/elements/1.1/source   http://purl.org/dc/elements/1.1/title   http://purl.org/dc/terms/description   
          Most people with Parkinson's disease have idiopathic Parkinson's disease (having no specific known cause). A small proportion of cases, however, can be attributed to known genetic factors. Mutations in specific genes have been conclusively shown to cause PD. These genes code for alpha-synuclein (SNCA), parkin (PRKN), leucine-rich repeat kinase 2 (LRRK2 or dardarin), PTEN-induced putative kinase 1 (PINK1), DJ-1 and ATP13A2.[4][22] In most cases, people with these mutations will develop PD. With the exception of LRRK2, however, they account for only a small minority of cases of PD.[4] The most extensively studied PD-related genes are SNCA and LRRK2. Mutations in genes including SNCA, LRRK2 and glucocerebrosidase (GBA) have been found to be risk factors for sporadic PD. The role of the SNCA gene is important in PD because the alpha-synuclein protein is the main component of Lewy bodies.[22] Missense mutations of the gene (in which a single nucleotide is changed), and duplications and triplications of the locus containing it have been found in different groups with familial PD. Mutations in LRRK2 are the most common known cause of familial and sporadic PD, accounting for approximately 5% of individuals with a family history of the disease and 3% of sporadic cases.
This is an updated copy of the human parkinson disease pathway (WP2371).
Sources: [wikipedia](http://en.wikipedia.org/wiki/Parkinson's_disease), [Qiagen](https://www.qiagen.com/geneglobe/pathwayview.aspx?pathwayID=345), and [KEGG](http://www.genome.jp/kegg/pathway/hsa/hsa05012.html). 
         http://purl.org/dc/terms/identifier   http://purl.org/dc/terms/references   
          https://identifiers.org/pubmed/15952880 
          https://identifiers.org/pubmed/15651302 
          https://identifiers.org/pubmed/18426756 
          https://identifiers.org/pubmed/17911161 
          https://identifiers.org/pubmed/16022590 
         http://purl.org/spar/cito/cites   
          https://identifiers.org/pubmed/23383003 
          https://identifiers.org/pubmed/19088304 
          https://identifiers.org/pubmed/15054086 
          https://identifiers.org/pubmed/15503154 
          https://identifiers.org/pubmed/15505146 
          https://identifiers.org/pubmed/18426756 
          https://identifiers.org/pubmed/15651302 
          https://identifiers.org/pubmed/15952880 
          https://identifiers.org/pubmed/16022590 
          https://identifiers.org/pubmed/17911161